At a Glance
Pes Planus (Flat Foot)
Collapse of the medial longitudinal arch
Adult Acquired Flatfoot
Most commonly from posterior tibial tendon dysfunction (PTTD)
Prevalence
~20–30% of adults have some degree of flat foot[5]
Key Concern
Untreated PTTD can progress from tendinitis to rigid deformity

Understanding the Collapsing Arch

The medial longitudinal arch is not a rigid structure but a dynamic system maintained by the interplay of bony architecture, plantar ligaments (especially the spring ligament), the plantar fascia, and the muscles of the foot and lower leg — chief among them the tibialis posterior muscle. When this system is overwhelmed, the arch collapses, the heel everts into valgus, and the forefoot abducts — producing the classic "too many toes" sign when viewing the foot from behind. This malalignment is not confined to the foot: it alters the biomechanics of the entire lower extremity, contributing to knee, hip, and even low back pain.

Posterior tibial tendon dysfunction (PTTD) is the most important and underrecognized cause of adult-acquired flatfoot. The posterior tibial tendon — the primary dynamic stabilizer of the arch — is prone to degenerative tendinopathy as it courses around the medial malleolus, an area of relatively poor vascularity. If untreated, PTTD progresses through four stages: from tendinitis with a supple flat foot, through progressive arch collapse with ligamentous failure, to a rigid, arthritic deformity that may require complex reconstructive surgery. The key clinical insight is that stage I and II PTTD are highly amenable to conservative care — but the window closes as the deformity becomes rigid.

Dr. Santopietro has extensive experience evaluating and managing the full spectrum of flat foot pathology — from the asymptomatic flexible flat foot in a child or adult who simply needs appropriate shoe support, to the adult with early PTTD who requires a custom University of California Biomechanics Laboratory (UCBL) device or articulated ankle-foot orthosis (AFO) to prevent progression. The custom orthotic prescription is tailored to the stage of disease, the patient's activity level, and their anatomy.

"The critical question is not 'do you have flat feet?' — many people with flat feet are pain-free and need no treatment. The question is: are your flat feet symptomatic, and are they progressing? That distinction requires a thorough biomechanical examination, not just a visual inspection."
Recognizing the Problem
Signs & Symptoms
Medial Arch Pain
Aching or burning along the inner arch, worsened by prolonged standing or walking; the most common presentation.
Heel Valgus
The heel tilts outward when standing; visible "rolling in" of the ankles is characteristic of arch collapse.
Tibialis Posterior Tenderness
Pain and swelling along the course of the posterior tibial tendon behind and below the medial malleolus (inner ankle).
Progressive Arch Flattening
The arch visibly collapses or has worsened over months to years; shoes wear unevenly on the inner edge.
Difficulty Standing on Tiptoe
Inability to perform a single-leg heel rise (standing on toes on one foot) is a key clinical sign of PTTD.
Knee & Hip Pain
Altered lower-limb alignment from flat feet commonly generates secondary knee (patellofemoral) and hip pain.
Why It Happens
Causes & Risk Factors
  • Posterior tibial tendon degeneration (most common cause of adult acquired flatfoot)
  • Ligamentous laxity — hypermobility of the spring ligament and plantar fascia
  • Hereditary flexible flat foot (congenital arch hypermobility)
  • Obesity placing excessive load on arch-supporting structures
  • Inflammatory arthritis (rheumatoid, seronegative spondyloarthropathy)
  • Neuromuscular conditions reducing intrinsic foot muscle strength
  • Tarsal coalition (bony bar between foot bones) — important in adolescents and young adults
  • Aging — progressive loss of tendon strength and ligamentous elasticity
Risk Factors
  • Age 40–70 (peak incidence of PTTD)
  • Women are affected more commonly than men[1]
  • Obesity (BMI > 30)
  • Hypertension and diabetes — both associated with PTTD
  • Prior medial ankle trauma or tendon injury
  • Prolonged corticosteroid use
  • Family history of hypermobile flat foot
Our Approach
Treatment & Management
1
Stage-Specific Evaluation
Dr. Santopietro determines whether the flat foot is flexible or rigid, and if PTTD is present, what stage. This drives the entire treatment approach — early stages are managed conservatively; late-stage rigid deformity is referred for surgical consultation.
2
Custom Foot Orthoses
For flexible flat foot and stage I–II PTTD, custom molded orthotics are the centerpiece of care. Devices are designed to support the medial arch, control hindfoot valgus, and reduce the load on the posterior tibial tendon. UCBL devices provide maximum control for severe presentations.
3
Physical Therapy & Strengthening
Tibialis posterior strengthening with eccentric and concentric protocols, combined with intrinsic foot muscle exercises and calf stretching, addresses the muscular component of arch support.[2]
4
Supportive Footwear
Motion-control or stability shoes with a firm heel counter and medial post significantly supplement orthotic support. Unsupportive footwear undermines even the best orthotic.
5
Activity Modification
High-impact activities are modified during active phases. Cross-training with low-impact exercise (cycling, swimming) maintains fitness while reducing tendon stress.
6
Bracing for Advanced Cases
Stage II PTTD with significant deformity may benefit from a custom-molded ankle-foot orthosis (AFO) or an Arizona brace for daily ambulation while conservative care proceeds.
Related Diagnoses

Also Diagnosed & Treated

Other conditions in this category that Dr. Santopietro regularly evaluates and treats.

Tibialis Posterior Tendonitis

The clinical term for the early inflammatory stage of posterior tibial tendon dysfunction (PTTD) — the same condition discussed throughout this page. When the posterior tibial tendon becomes irritated and inflamed before structural deformity has set in, targeted rehabilitation and custom orthotics are highly effective at halting progression.

Tarsal Coalition

A congenital fusion — bony, cartilaginous, or fibrous — between two or more tarsal bones that produces a rigid or semi-rigid flatfoot. Most commonly involves the calcaneonavicular or talocalcaneal joint. Presents with recurrent ankle sprains, peroneal spasm, and a foot that cannot be fully corrected passively. Evaluation and conservative management are appropriate before considering surgical resection.

Morton's Neuroma

A perineural fibrosis of the interdigital nerve, most commonly between the third and fourth metatarsal heads. Produces burning, shooting, or electric-shock pain in the forefoot and toes, often with a sensation of walking on a pebble. Biomechanical factors — particularly hypermobility and forefoot splay associated with flat feet — increase nerve compression, and metatarsal padding or orthotic support frequently reduces symptoms.

Evidence Base
Clinical References
Kohls-Gatzoulis J, Angel JC, Singh D, Haddad F, Livingstone J, Berry G. "Tibialis posterior dysfunction: a common and treatable cause of adult acquired flatfoot." BMJ. 2004;329(7478):1328–1333.
Kulig K, Reischl SF, Pomrantz AB, et al. "Nonsurgical management of posterior tibial tendon dysfunction with orthoses and resistive exercise." Phys Ther. 2009;89(1):26–37.
Durrant B, Chockalingam N, Hashmi F. "Posterior tibial tendon dysfunction: a review." J Am Podiatr Med Assoc. 2011;101(2):176–186.
Myerson MS. "Adult acquired flatfoot deformity: treatment of dysfunction of the posterior tibial tendon." J Bone Joint Surg Am. 1996;78(5):780–792.
Staheli LT, Chew DE, Corbett M. "The longitudinal arch: a survey of eight hundred and eighty-two feet in normal children and adults." J Bone Joint Surg Am. 1987;69(3):426–428.

Flat Feet Don't Have to Mean Chronic Pain

With the right orthotic prescription and a biomechanically sound treatment plan, most patients with flat feet and arch pain can return to full, pain-free activity.

Book an Appointment Call 617.734.0003